ناخذ كمثال غاز ال vx اقرا بعض المعلومات عنه و عن الترياق الخاص به و اخبرنى ان كان يوجد فى مكونات التمر
With its high viscosity and low volatility, VX has the texture and feel of high-grade motor oil. This makes it especially dangerous, as it has a high persistence in the environment. It is odourless and tasteless, and can be distributed as a liquid or, through evaporation, into small amounts of vapour. It works as a nerve agent by blocking the function of the enzyme acetylcholinesterase. Normally, an electric nerve pulse would cause the release of acetylcholine over a synapse that would stimulate muscle contraction. The acetylcholine is then broken down to non-reactive substances (acetic acid and choline) by the acetylcholinesterase enzyme. If more muscle tension is needed the nerve must release more acetylcholine. VX blocks the action of acetylcholinesterase, thus resulting in sustained contractions of all the muscles in the body. Sustained contraction of the diaphragm muscle causes death by asphyxiation.
As little as 200 micrograms is enough to kill an average person, depending on method of absorption. If the absorbed dose is not too high, death can be avoided if the appropriate antidote is injected immediately after exposure. The most commonly used antidotes are atropine, pralidoxime, and diazepam which are, in several nations, issued for military personnel in the form of an autoinjector. Atropine works by binding and blocking a subset of acetylcholine receptors (known as muscarinic acetylcholine receptor, mAchR), so that the build up of acetylcholine produced by loss of the acetylcholinesterase function can no longer affect their target. The injection of pralidoxime regenerates bound acetylcholinesterase.
و اذن اين الانتى دوت هنا فى التمر ؟؟
و على هذا المثال يمكنك القياس لمئات السموم الغازية و الكيميائية
With its high viscosity and low volatility, VX has the texture and feel of high-grade motor oil. This makes it especially dangerous, as it has a high persistence in the environment. It is odourless and tasteless, and can be distributed as a liquid or, through evaporation, into small amounts of vapour. It works as a nerve agent by blocking the function of the enzyme acetylcholinesterase. Normally, an electric nerve pulse would cause the release of acetylcholine over a synapse that would stimulate muscle contraction. The acetylcholine is then broken down to non-reactive substances (acetic acid and choline) by the acetylcholinesterase enzyme. If more muscle tension is needed the nerve must release more acetylcholine. VX blocks the action of acetylcholinesterase, thus resulting in sustained contractions of all the muscles in the body. Sustained contraction of the diaphragm muscle causes death by asphyxiation.
As little as 200 micrograms is enough to kill an average person, depending on method of absorption. If the absorbed dose is not too high, death can be avoided if the appropriate antidote is injected immediately after exposure. The most commonly used antidotes are atropine, pralidoxime, and diazepam which are, in several nations, issued for military personnel in the form of an autoinjector. Atropine works by binding and blocking a subset of acetylcholine receptors (known as muscarinic acetylcholine receptor, mAchR), so that the build up of acetylcholine produced by loss of the acetylcholinesterase function can no longer affect their target. The injection of pralidoxime regenerates bound acetylcholinesterase.
و اذن اين الانتى دوت هنا فى التمر ؟؟
و على هذا المثال يمكنك القياس لمئات السموم الغازية و الكيميائية


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